Post-lecture activity (Week 10)

OBRA - POST-LECTURE ACTIVITY (WEEK 10)

OBRA - POST-LECTURE ACTIVITY (WEEK 10)

by Jess Dominic Obra -
Number of replies: 1

Adequate cardiac output is necessary in order to supply oxygen and other nutrients to all body tissues. Cardiac output is influenced by stroke volume and heart rate. Factors affecting the stroke volume include PRELOAD, CONTRACTILITY and AFTERLOAD.

In the following conditions, indicate what factor/s influence the cardiac output: PRELOAD, CONTRACTILITY, AFTERLOAD, HEART RATE. Explain why the factor/s affects cardiac output in 1 to 2 sentences.

a. High blood pressure

Any element that boosts heart rate, stroke volume, or both in order to increase cardiac output will raise blood pressure and encourage blood flow. Thus, premised on this scenario, less elastic, narrow arteries will make it more challenging for blood to circulate effectively throughout the body (American Heart Association, 2022).

b. Massive bleeding

Acute blood loss reduces blood volume, which lowers central venous pressure and decreases heart filling. As a result, cardiac output and arterial pressure are decreased (Klabunde, 2021).

c. The drug dopamine

Unlike other inotropic drugs, Dopamine is a direct-acting catecholamine that yields various benefits for treating visceral hypoperfusion conditions including shock and refractory heart failure for the particular reason that it enhances myocardial contractility. Therefore, the inception of such a phenomena would boil down to an increased stroke volume, heart rate, and cardiac output (Reid & Thompson, 1975).

d. Running a sprint

The intersection between the pulmonary ventilation and cellular respiration lies in the  cardiovascular system as the heart must pump blood at a sufficient velocity to ensure an adequate and continuous supply of oxygen (Evans, 1985). Owing to this ground, workload will cause a proportional rise in heart rate when running.

e. Hyperthyroidism with increased secretion of thyroid hormones

The most frequent cause of hyperkinetic circulatory illness is hyperthyroidism. Along with its cellular effects, hyperthyroidism also affects the heart's hemodynamics, increasing preload and contractility while lowering systemic vascular resistance raises cardiac output (Osuna et al., 2017).

f. Massive myocardial infarction (cardiac tissue death)

Cardiac function is compromised by the loss of viable myocardium in the above mentioned case.  Such incidents can result in decreased cardiac output and, in extreme cases, cardiogenic shock (Mechanic et al., 2022).

g. Hypothermia

A reflex adrenergic vasoconstriction of the skin (noradrenalin) occurs when the core temperature is reduced (hypothermia). As bradycardia increases, cardiac output decreases to below a core temperature of 34 degrees Celsius (Deussen, 2007).

h. Emotional distress

Emotional tension sets off a chain reaction in the body and in the process, stress hormones namely cortisol and adrenaline are released. This triggers the heart to beat more quickly, makes blood arteries constrict, and also accentuates the intensity of blood pressure (Torpy et al., 2007).

In reply to Jess Dominic Obra

Re: OBRA - POST-LECTURE ACTIVITY (WEEK 10)

by Jess Dominic Obra -
References:

American Heart Association (2022). How High Blood Pressure Can Lead to Heart Failure. https://www.heart.org/en/health-topics/high-blood-pressure/health-threats-from-high-blood-pressure/how-high-blood-pressure-can-lead-to-heart-failure

Deussen, A. (2007). [Hyperthermia and hypothermia. Effects on the cardiovascular system]. https://pubmed.ncbi.nlm.nih.gov/17554514/

Evans DL. Cardiovascular adaptations to exercise and training. Vet Clin North Am Equine Pract. 1985 Dec;1(3):513-31. doi: 10.1016/s0749-0739(17)30748-4. PMID: 3877552.

Klabunde, R. (2021). Cardiovascular Physiology Concepts. https://www.cvphysiology.com/Blood%20Pressure/BP031

Mechanic et al. (2022). Acute Myocardial Infarction. https://www.ncbi.nlm.nih.gov/books/NBK459269/

Osuna PM, Udovcic M, Sharma MD. Hyperthyroidism and the Heart. Methodist Debakey Cardiovasc J. 2017 Apr-Jun;13(2):60-63. doi: 10.14797/mdcj-13-2-60. PMID: 28740583; PMCID: PMC5512680.

Reid PR, Thompson WL. The clinical use of dopamine in the treatment of shock. Johns Hopkins Med J. 1975 Dec;137(6):276-9. PMID: 1563.

Torpy JM, Lynm C, Glass RM. Chronic Stress and the Heart. JAMA. 2007;298(14):1722. doi:10.1001/jama.298.14.1722